2005). These success weren't surprising if we think about that PEA is really a improperly water?soluble compound, which may limit its oral absorption and bioavailability, However they might also suggest a short?lived motion of PEA, in settlement with The point that this compound is degraded
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?acylethanolamines (NAEs), endogenous biologically Energetic lipids such as the endog
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PEA protects in opposition to bacterial an infection via innate immune modulation involving MCs, macrophages and microglia. Prophylactic PEA at a dose of 0.1 mg/kg body body weight prolonged survival fee and minimized neuro-inflammation in an aged murine bacterial meningitis design, from the abs